产品名称:单克隆抗体,Anti-Methamphetamine(4D2)抗体
中文名称:单克隆抗体抗体
英文名称:Anti-Methamphetamine(4D2)
产品编号:byk-2068M
产品规格:0.1ml/0.2ml
产品用途:科研实验
性 状: Lyophilized or Liquid
浓 度: 1mg/1ml
亚 型: IgG
贮 存: 贮存于-20℃.
相关标记: HRP Biotin Gold RBITC AP FITC Cy3 Cy5 Cy5.5 Cy7 PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 APC Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647
我公司拥有领先的细胞服务技术,先进的仪器设备和专业的ELISA检测试剂盒、生物索标记、荧光索标记、酶标记等,广泛用于多种分析研究与技术测定。欢迎来电咨询,我们将竭诚为你服务!
单克隆抗体,Anti-Methamphetamine(4D2)抗体 相关产品:
磷酸化丝裂原活化蛋白激酶激酶1抗体 Anti-phospho-MEK1/MAPKK1(Ser298)
丝裂原活化蛋白激酶1抗体 Anti-ERK2/MAPK1
β氨基己糖苷酶A抗体 Anti-HEXA/Beta hexosaminidase subunit alpha
DNA切除修复蛋白1抗体 Anti-ERCC1
内质网Aβ相关结合蛋白抗体 Anti-ERAB/HSD17B10
雌激素受体α抗体 Anti-Estrogen Receptor alpha
酪氨酸蛋白激酶受体B6抗体 Anti-EphB6
障碍自整合蛋白BAF抗体 Anti-BANF1/BAF
蛙皮降压肽抗体 Anti-Sauvagine
红细胞生成素受体抗体 Anti-EPOR
酪氨酸蛋白激酶受体B2抗体 Anti-EphB2 R
酪氨酸蛋白激酶A4受体抗体 Anti-EphA4 R/Eph receptor A4
丝氨酸/苏氨酸蛋白激酶DCAMKL1抗体 Anti-DCAMKL1
乙酰肝素酶2抗体 Anti-Heparanase 2
内皮细胞受体蛋白酪氨酸激酶抗体 Anti-EphA2
视神经萎缩相关蛋白1抗体 Anti-OPA1
酪氨酸蛋白激酶A4抗体 Anti-EPH receptor A4
脑磺基转移酶样蛋白4A1抗体 Anti-SULT4A1
内皮细胞受体蛋白酪氨酸激酶A抗体 Anti-EphA1/Ephrina1/Ephrin A1
肿瘤坏死因子α诱导蛋白4抗体 Anti-EphA1/Ephrina1/Ephrin A1
抗体规律
(1)初次反应产生抗体:当抗原一次进入机体时,需经一定的潜伏期才能产生抗体,且抗体产生的量也不多,在体内维持的时间也较短。
(2)再次反应产生抗体:当相同抗原第二次进入机体后,开始时,由于原有抗体中的一部分与再次进入的抗原结合,可使原有抗体量略为降低。随后,抗体效价迅速大量增加,可比初次反应产生的多几倍到几十倍,在体内留存的时间亦较长。
(3)回忆反应产生抗体:由抗原刺激机体产生的抗体,经过一定时间后可逐渐消失。此时若再次接触抗原,可使已消失的抗体快速上升。如再次刺激机体的抗原与初次相同,则称为特异性回忆反应;若与初次反应不同,则称为非特异性回忆反应。非特异性回忆反应引起的抗体的上升是暂时性的,短时间内即很快下降。
单克隆抗体,Anti-Methamphetamine(4D2)抗体 产品详情:
Methamphetamine (METH) is closely related chemically to amphetamine (AMPH). METH is a potent central nervous system stimulant with additional peripheral sympathomimetic effects. METH and AMPH have been used clinically in the treatment of obesity, minimal brain dysfunction, narcolepsy, depression and to counter fatigue. They are also subjected to widespread abuse. METH is an indirect agonists. It causes the release of newly synthesized norepinephrine and dopamine and it blocks the re uptake of these transmitters from the synapse. This can lead to an increase in the concentration of catecholamines in the synapse as well as an overall increase in catecholaminergic activity in the brain. The mechanism of METH induced neurotoxicity for all monoaminergic cell types may lie primarily with the dopaminergic system in the striatum. It may also lie with the interaction between METH induced release of dopamine and its ability to inhibit monoamine oxidase.