Anti-HCV Core protein抗体,丙肝病毒核心抗原C区抗体实实在在

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产品名称：Anti-HCV Core protein抗体,丙肝病毒核心抗原C区抗体实实在在
中文名称：丙肝病毒核心抗原C区抗体
英文名称：Anti-HCV Core protein
产品编号：BYK-0221R
产品别名：Core protein p19; Hepatitis C Virus Core Antigen-C; HCVAg; HCV-Core protein;HCV core antigen; HCV core protein; Hepatitis C Virus core protein; capsid protein; polyprotein precursor; Core protein p21; Core protein p9.

本公司另供应“丙肝病毒核心抗原C区抗体”的标记有：Alexa Fluor 350 Alexa Fluor 488 Alexa Fluor 555 Alexa Fluor 647 AP APC Biotin Cy3 Cy5 Cy5.5 Cy7 FITC Gold HRP PE PE-Cy3 PE-CY5 PE-CY5.5 PE-CY7 RBITC.
产品规格：100ug/200ug
产品浓度： 1mg/1ml
抗体来源：兔源、鼠源、山羊
克 隆：单克隆抗体、多克隆抗体
抗体类型：一抗
性 状： 冻干粉或液体
保存条件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-HCV Core protein抗体,丙肝病毒核心抗原C区抗体实实在在技术说明，产品推荐，由于篇幅原因，更多信息请：

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Anti-E.coli O6 （Escherichia coli O6; EPEC O6）  抗大肠埃希杆菌O6抗体（肠致病性大肠杆菌O6）
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产品介绍：

Core protein packages viral RNA to form a viral nucleocapsid, and promotes virion budding. Modulates viral translation initiation by interacting with HCV IRES and 40S ribosomal subunit. Also regulates many host cellular functions such as signaling pathways and apoptosis. Prevents the establishment of cellular antiviral state by blocking the interferon-alpha/beta （IFN-alpha/beta） and IFN-gamma signaling pathways and by inducing human STAT1 degradation. Plays an important role in virus-mediated cell transformation leading to hepatocellular carcinomas. Interacts with, and activates STAT3 leading to cellular transformation. May repress the promoter of p53, and sequester CREB3 and SP110 isoform3/Sp110b in the cytoplasm. Also represses cell cycle negative regulating factor CDKN1A, thereby interrupting an important check point of normal cell cycle regulation. Targets transcription factors involved in the regulation of inflammatory responses and in the immune response: suppresses NK-kappaB activation, and activates AP-1. Mediates apoptotic pathways throught association with TNF-type receptors TNFRSF1A and LTBR, although its effect on death receptors-induced apoptosis remains controvertial. Enhances TRAIL mediated apoptosis, suggesting that it might play a role in mediated apoptosis, suggesting that it might play a role in immune-mediated liver cell injury. Secreted core protein is able to bind C1QR1 at the T-cell surface, resulting in down-regulation of T-lymphocytes proliferation. May transactivate human MYC, Rous sarcoma virus LTR, and SV40 promoters. May suppress the human FOS and HIV-1 LTR activity. May alter lipid metabolism by interacting with hepatocellular proteins involved in lipid accumulation and storage.