磷酸化脊髓小脑失调症蛋白1抗体,Ataxin 1（Ser775）抗体

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抗体名称：磷酸化脊髓小脑失调症蛋白1抗体,Ataxin 1（Ser775）抗体

产品编号：BYK-12534R

别 名：Ataxin 1 （phospho S776）;p-Ataxin 1 （phospho S776）; ATX1; ATXN1; SCA1; Ataxin 1; Ataxin-1; Ataxin1; Spinocerebellar ataxia type 1; ATX1_HUMAN.

相关标记：Alexa Fluor 350   Alexa Fluor 488   Alexa Fluor 555   Alexa Fluor 647   AP   APC Biotin   Cy3   Cy5   Cy5.5   Cy7    FITC    Gold    HRP    PE   PE-Cy3    PE-CY5    PE-CY5.5   PE-CY7     RBITC.

浓 度： 1mg/1ml
抗体来源（宿主）：兔源、鼠源、其他
克隆：单克隆抗体、多克隆抗体
适用：Human, Mouse, Rat, Chicken, Dog, Pig, Cow, Horse, Sheep, Monkey, others。 （电询）
抗体类型：一抗
性 状： 冻干粉或液体
储 存 液： Preservative: 15mM Sodium Azide, Constituents: 1% BSA, 0.01M PBS, pH 7.4 or PBS with 0.1% sodium azide and 50% glycerol pH 7.3. -20oC, Avoid freeze / thaw cycles.
产品应用 Immunohistochemistry （IHC）, Flow Cytometry （FACS） , Western Blotting （WB） , ELISA , Immunohistochemistry , Immunohistochemistry （Paraffin-embedded Sections） （IHC （p）） , Immunoprecipitation （IP） , Immunocytochemistry （ICC） ，Immunofluorescence （IF）等。（咨询，点击查看）

保存条件：Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

磷酸化脊髓小脑失调症蛋白1抗体,Ataxin 1（Ser775）抗体产品介绍：Ataxin-1, also designated spinocerebellar ataxia type 1 protein （Sca-1）, is differentially expressed and localizes to both the cytoplasm and the nucleus. Mutations in Ataxin-1 are associated with the onset of the autosomal dominant neurodegenerative disorder spinocerebellar ataxia type 1 （SCA-1）, which is characterized by progressive neuronal loss in the cerebellum, muscle wasting and ataxia. In Purkinje cells, where SCA-1 is predominantly observed, Ataxin-1 has been shown to directly associate with the Purkinje-enriched leucine-rich acidic nuclear protein （LANP） and the nuclear matrix-associated protein promyelocytic leukemia protein PML. In SCA-1, Ataxin-1 is mutated to encode a polyglutamine protein that forms nuclear aggregates, which interact significantly more strongly with LANP and contribute to the pathogenesis of SCA-1.

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